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Stanford AIMI 61

Stable right basilar opacity and small right pleural effusion with minimal improvement in aeration of the left lung base.

Mild prominence and indistinctness of the hila, possibly due to mild vascular congestion.

Mediastinal and hilar contours are normal and unchanged

Cardiac silhouette appears normal; however, there are indications of mild pulmonary edema.

Persistent right upper lobe opacity, which may represent residual airspace disease or edema.

Interval slightly decreased lung volumes. No focal consolidation.

Presence of a dual-lead AICD pacemaker.

Ill-defined airspace opacity within the left lower lobe on both frontal and lateral views, suggestive of a developing infectious process, with aspiration less likely. No significant pleural effusion noted.

Successful placement of a left subclavian central venous catheter with the tip in the SVC.

No significant interval change with stable cardiomegaly, mild interstitial pulmonary edema, retrocardiac opacity, and a small left pleural effusion.

Presence of a known right diaphragmatic hernia.

Signs suggestive of pulmonary edema, as indicated by the increased cardiothoracic index and changes in the pulmonary hila and parahilar regions.

A 5 mm nodular opacity in the right upper lung zone, likely representing a calcified granuloma.

Left PICC line terminating at the level of the trachea.

Chest tube in position on the left side.

Diffuse bilateral pulmonary parenchymal involvement with a predominance in the left hemithorax and left superior division.

Accentuated kyphosis observed.

No evidence of adenopathy; previously noted adenopathy has resolved.

Right-sided subclavian line with tip in the mid SVC.

Presence of pleural calcifications along the left mid thorax.

Increased dense consolidation within the left lower lobe, which may represent atelectasis and/or consolidation.

Distended esophagus unchanged.

Persistent linear atelectasis in the left lower lobe.

Presence of multiple lines and tubes with appropriate positioning.

Mediastinal and hilar adenopathy, with recommendation for CT evaluation.

Stable post-surgical changes of coronary artery bypass grafting.

Increased pulmonary markings observed.

New right lower lobe opacity, possibly indicative of pneumonia; further oblique views recommended for evaluation.

Stent graft in the proximal to mid descending thoracic aorta with stable positioning of endotracheal tube, right-sided chest tube, and nasogastric tube.

Additional right internal jugular catheter.

Degenerative changes and osteophytes present along the thoracic spine.

No significant change, structures are stable.

Endotracheal tube with tip positioned 3 cm above the carina.

Dual lead pacer wires present on the left, with leads intact and in position; no attached generator observed.

Postsurgical changes consistent with recent median sternotomy, including the placement of a right IJ CVC, Swan-Ganz catheter, mitral valvuloplasty, bioprosthetic aortic valve, mediastinal drains, epicardial pacing wires, and sternotomy wires.

12 mm faint nodular density in the right lower lung, raising the possibility of focal parenchymal disease or pulmonary nodule. Further evaluation with follow-up PA and lateral chest radiographic examination and possibly CT examination of the thorax is recommended.

Right infrahilar confluence suggesting possible pulmonary edema.

Nasogastric tube in place, extending beyond the gastroesophageal junction.

Stable positioning of lines and tubes from previous imaging.

Left basal subsegmental atelectasis without additional pulmonary abnormalities.

Presence of right internal jugular tunneled catheter and left lower hemithorax pigtail drain.

Mildly enlarged cardiac silhouette with prosthetic valve, indicative of underlying cardiac pathology.

Slight prominence in the region of the AP window, possibly representing a slightly prominent pulmonary artery; differential includes lymphadenopathy

Pacemaker present in the left axilla with leads in the right atrium and right ventricle, unchanged

Decreased size of previously seen right apical pneumothorax with a tiny residual pneumothorax remaining.

Indistinct vasculature and patchy hazy opacities suggesting moderate pulmonary edema.

Chronic cardiomegaly and biventricular decompensation without current evidence of pulmonary edema.

Left ventriculoperitoneal shunt present.

New opacity at the left base, concerning for pneumonia.

Unchanged bibasilar opacity, more pronounced on the left, which may represent atelectasis, consolidation, or aspiration.

Stable mediastinal and cardiac shift to the right.

The lungs are clear without evidence of pulmonary edema or pleural effusion. The cardiomediastinal silhouette is normal in size and appearance. Osseous structures of the chest are unremarkable.

The left lower lobe mass is not visualized due to superimposition with the cardiac silhouette.

Nasogastric tube within the esophagus; further advancement may be necessary.

Persistent right lower lobe collapse with loss of the right heart border and some mediastinal shift to the right. The right-sided pleural effusion has markedly decreased since the prior study post-thoracentesis. There is a slight increase in the left-sided pleural effusion.

Endotracheal tube terminates 2.3 cm from the carina

Left apical pneumothorax measuring 5 cm, persistent and unchanged.

Bilateral pulmonary consolidations in the upper middle fields, likely of infectious etiology.

High-density nodule at the level of the lingula, likely representing a granuloma.

Bilateral calcified pleural plaques suggesting prior asbestos exposure.

Limited evaluation of lateral views due to respiratory motion.

Prominent aortic arch without significant change, likely related to imaging technique.

Partially visualized stent in the abdomen.

Nodular densities in the left apex, measuring 2.5 cm and 1.2 cm

Interval placement of 2 right-sided chest tubes.

Additional catheter seen overlying the right infraclavicular region, uncertain if external; recommend clinical correlation.

Multiple foreign bodies within the soft tissues of the right shoulder and upper back, likely representing shotgun pellets.

Bilateral basal bronchiectasis, which could be associated with chronic pulmonary disease.

Slight increase in AP diameter with mild flattening of the hemidiaphragms, suggestive of emphysema.

Increased opacity at the lung bases, likely due to atelectasis secondary to low lung volumes.

Stable heart size and mediastinal contour with the presence of sternal wires and mediastinal clips, indicative of prior heart/lung transplant.

Right internal jugular central venous catheter in unchanged appropriate position.

Interval improvement in the indistinctness of the pulmonary vasculature, suggesting a decrease in pulmonary edema.

Evidence of thoracic spine spondylosis.

Relative improvement in the size of the right pneumothorax when compared to the unspecified previous study.

Multiple bilateral nodular opacities throughout both lungs.

Normal cardiomediastinal silhouette for patient's lung volumes and radiographic technique.

Very low lung volumes with increased retrocardiac opacity, which is better seen on the lateral view, concerning for pneumonia. Clinical correlation is recommended.

Slight improvement in right upper lobe consolidation in the posterior segment.

Tracheostomy cannula is appropriately placed 4 cm from the carina.

Persistent right subcutaneous emphysema, unchanged from prior examination.

Nasogastric tube in situ with the tip projecting within the stomach.

Moderate elevation of the left diaphragm, possibly related to moderate gas-distended stomach with fluid level.

Difficulty in assessing pulmonary vascularity due to extensive pulmonary changes, with probable vascular engorgement and enlargement of the cardiac silhouette suggestive of volume overload.

Possible ground glass opacity in the middle and left lung fields adjacent to the lower scapular region.

Endovascular prosthesis in the abdominal aorta.

Bibasilar and perihilar airspace opacities

Presence of a small right pleural effusion with pleural thickening.

Metallic stent in the region of the inferior vena cava (IVC) is present.

Single left chest tube in situ.

Left shoulder findings require dedicated shoulder radiograph for detailed evaluation.

Increased large left pleural effusion with adjacent compressive atelectasis, underlying mass not excluded.

Low lung volumes with central pulmonary vascular congestion.

NG tube in satisfactory position within the stomach.

Right internal jugular central catheter with its tip near the cavoatrial junction.

Persistent enlargement of the pulmonary arterial contour suggestive of pulmonary hypertension or volume overload.

Lung fields are well inflated with no evidence of significant pneumothorax on the chest X-ray.

Widening of the right acromioclavicular joint, unchanged from prior.

Mildly enlarged heart size with mild left and right ventricular enlargement.

Stable position of TIPS.