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Stanford AIMI 60

Bilateral opacities throughout the lungs consistent with moderate pulmonary edema.

Small bilateral pleural effusions are present with adjacent compressive atelectasis.

Impression on the lower cervical trachea may indicate the presence of a right thyroid mass.

The left lung is well expanded with no new areas of consolidation.

Hyperlucency of peripheral vessels in the left hemithorax, likely representing residual changes from a previous infectious/inflammatory process.

Unchanged position of the left PICC line and esophageal stent.

Interval development of moderate pulmonary edema, with the overall stable appearance of bibasilar opacities and small bilateral pleural effusions.

Cardiothoracic index is at the upper limit of normal, with no overt cardiomegaly.

Clear lungs with no new pulmonary opacities.

Abnormal increased opacity in both lung bases, more pronounced on the left.

Increased left pleural effusion and left lower lung opacity suggestive of atelectasis or consolidation.

Dialysis catheter is appropriately positioned with its tip in the upper right atrium.

Endotracheal tube, enteric tube, and right subclavian central venous line have been removed.

Atrial lead termination appears medially, suggesting potential malposition.

Improved aeration of the left lung, though basilar airspace opacity and pleural effusion persist.

Unchanged position of sternotomy wires, surgical clips, and left upper extremity peripherally inserted central catheter (PICC line).

Prominence of upper lobe vessels suggesting pulmonary venous hypertension.

Interval increase in bibasilar opacities, suggestive of atelectasis or early consolidation.

Normal heart size and no evidence of pneumothorax or large pleural effusion.

Mediastinal widening is noted, likely related to postsurgical changes and presence of drains.

Prosthetic aortic valve visualized

Presence of endotracheal and nasogastric tubes, with the latter's tip not visualized.

Persistent reduced lung volumes with new findings suggestive of mild interstitial pulmonary edema.

Prominent mediastinum consistent with known mediastinal mass.

Destruction and sclerosis of both glenohumeral joints, suggestive of rheumatoid arthritis.

Increased bilateral pulmonary opacifications likely representing pulmonary edema.

Stable straightening of lumbar lordosis with preserved spinal alignment and intervertebral disc spaces.

Cardiomegaly with interstitial edema suggestive of congestive heart failure (CHF).

Persistent linear opacities in the left lower lobe retrocardiac region, likely representing scarring or atelectasis.

Thoracic aorta is enlarged but without focal aneurysmal dilation.

Chronic airlessness of the left lung.

Slightly increased elevation of the right hemidiaphragm, which may indicate true elevation or a subpulmonic effusion.

Presence of a nasogastric (NG) tube extending beneath the diaphragm, coiled over the stomach.

Stable diffuse irregular interstitial pattern, suggestive of a combination of emphysema and pulmonary edema.

Improvement in the size of bilateral pleural effusions, particularly on the right side.

Stable cardiomedialstinal silhouette and pulmonary vasculature without evidence of additional pleural effusions.

Unchanged position of left pigtail thoracostomy tube, terminating at the left lung base.

Persistent dense left basilar opacity suggestive of atelectasis, aspiration, or infection.

Mild cardiomegaly, unchanged from previous.

Other visualized bony structures of the chest appear unremarkable.

No evidence of acute pulmonary or pleural disease.

Prominent epicardial fat pad noted around the apex of the heart.

Mild bronchial wall thickening, which may indicate bronchitis.

Chronic volume loss in the left upper lobe with associated elevation of the left hemidiaphragm.

Hemidiaphragms are sharply defined with no evidence of acute focal pneumonia.

The distal tip of the right internal jugular (IJ) central line and right peripherally inserted central catheter (PICC) line are not well visualized.

Sharp posterior costophrenic angles, indicating resolution of previously seen effusions.

Nasogastric tube tip near the gastroesophageal junction

Loculated fluid in the right major fissure, no change

Patchy parenchymal opacities in both lung bases, concerning for infection.

Presence of a right internal jugular sheath and feeding tube, with the feeding tube coiled and its tip oriented towards the gastroesophageal junction.

Patchy mid to lower left lung opacity concerning for pneumonia; recommend follow-up to resolution to exclude an underlying pulmonary lesion.

Small left hydropneumothorax is unchanged.

Minimal right lung base atelectasis

Presence of left pleural drain.

Left basal opacity is similar to previous study.

Lung hyperinflation without acute pulmonary pathology.

Small bilateral pleural effusions with improvement noted.

Left internal jugular line terminating at the origin of the superior vena cava, and a nasogastric tube passing below the diaphragm without complications.

Bilateral peripheral patchy consolidations, suggestive of an infectious process, possibly COVID-19, in the appropriate clinical context.

Low lung volumes without focal pulmonary abnormality, aside from minimal bibasilar atelectasis.

Ill-defined bibasilar opacities likely representing atelectasis.

No evidence for pneumonia, edema, or pneumothorax.

Decreased subcutaneous emphysema along the right lower chest wall.

Stable small inferior left pneumothorax

Normal heart size. The aorta is elongated, consistent with prior imaging.

Nasoenteric catheter is appropriately positioned with the tip in the stomach.

No change from prior imaging.

Presence of mild atelectatic streaks at the left lung base.

Courses below the level of the diaphragm; inferior aspect not well visualized.

Vascular crowding, predominantly on the left.

Old right 7th rib fracture.

Minimal anterior wedging of mid-thoracic vertebral bodies with associated mild kyphosis.

Presence of re-expansion pulmonary edema at the right base.

Right internal jugular central venous catheter with its tip located in the mid superior vena cava.

Bilateral interstitial edema with patchy areas of consolidation consistent with edema and/or infection.

Slight improvement in small bilateral pleural effusions and associated bibasilar atelectasis.

There is no radiographic sign of pulmonary edema.

Streaky opacity at the right base, likely representing atelectasis

New marked bilateral perihilar opacities with numerous Kerley B lines, suggestive of flash pulmonary edema.

Normal pulmonary vascularity with heart size accentuated by shallow inspiration.

Right apical small pneumothorax with the chest tube in place medially.

Basilar opacities likely representing postoperative atelectasis, left greater than right.

Unchanged small left pleural effusion with low lung volumes; slight improvement in pulmonary edema.

Mildly prominent mediastinum, likely related to position and technique.

Bibasilar atelectasis, worse in the left lung.

New airspace opacities overlying the right lower lung.

Probable left upper lobe collapse.

Opacification in the lung bases on the frontal view raises concern for pneumonia; however, the absence of lateral view correlation favors atelectasis.

Exchange of left subclavian introducer for triple lumen catheter with tip positioned in the right atrium.

Right-sided central venous catheter in place, with the tip located in the mid to distal superior vena cava.

Stable appearance of mediastinal and cardiac contours.

Low lung volumes without definitive pulmonary pathology.

Healing distal clavicle fracture on the right side.

Unremarkable upper abdominal appearance.

Central venous catheter in situ via the right jugular vein.

Unchanged persistent left retrocardiac opacity, indicating stable cardiopulmonary status.

Persistent right apical pneumothorax, reduced in size compared to prior imaging.

Tip of central venous catheter in mid superior vena cava.

Mechanical changes in the spine, which could be due to degenerative disc disease or other chronic spinal conditions.