text: Endothelium-dependent relaxations, nitric oxide (NO) and endothelium derived hyperpolarizing factor (EDHF)-type, were studied in rabbit iliac artery and aortic rings using the G protein-coupled receptor agonist acetylcholine (ACh) and by cyclopiazonic acid (<< CPA >>), which promotes store-operated Ca(2+) entry by inhibiting the endothelial [[ SERCA ]] pump.